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A few repurposed agents have been proposed: Minocycline → anti-inflammatory, possible antiviral effects Ivermectin → broad in vitro antiviral activity However, it is important to be precise: Evidence in influenza remains limited or indirect These agents should be viewed as adjuncts , not foundational therapies Ivermectin’s antiviral effects appear to stem from a host-directed mechanism , not a direct virucidal action: It inhibits the importin α/β nuclear transport system Many RNA viruses (including influenza) rely on this pathway to shuttle viral proteins into the nucleus In influenza, viral ribonucleoproteins must enter the nucleus for replication Blocking this pathway can reduce viral replication in vitro This mechanism has been demonstrated across multiple RNA viruses (e.g., dengue, Zika, SARS-CoV-2), suggesting a broad but weak antiviral effect What About Tamiflu