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In summary, MSC- and exosome-based therapies mitigate OA ferroptosis through the METTL3–ACSL4, SNHG7–miR-485-5p–FSP1, and GOT1–CCR2–Nrf2 axes

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By contrast, reactive astrocytes modulate Ca 2+ signaling by overexpressing calcineurin, a Ca 2+ -dependent phosphatase that activates inflammation-related genes