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A number of research confirmed a strong involvement of oxidative stress in the pathophysiology of neurodegenerative diseases through a variety of mechanisms including induction of oxidation of macromolecules such as nucleic acids, proteins, lipids, mitochondrial dysfunction, glial cell activation, amyloid β deposition, apoptosis, and proteasome dysfunction [3, 5, 10, 11]
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Although many publications dating back to the beginning of this century about antioxidants in general (and GSH in particular) only tend to underline the beneficial effects that these substances have in cancer patients, possibly preventing cancer onset and curing anticancer treatment adverse events, much more has been discovered in recent years, as widely displayed in this review
